[1] ZHU C,WU Y P,JIANG Z Y,et al.Dietary soy isoflavone attenuated growth performance and intestinal barrier functions in weaned piglets challenged with lipopolysaccharide[J].International Immunopharmacol,2015,28(1):288-294.
[2] 陈少魁,刘玉兰,王海波,等.亚麻籽油对脂多糖刺激仔猪肝脏Toll样受体4和核苷酸结合寡聚化结构域信号通路关键基因表达的影响[J].动物营养学报,2016,28(3):891-898.
[3] LIU Y L,CHEN F,ODLE J,et al.Fish oil enhances intestinal integrity and inhibits TLR4 and NOD2 signaling pathways in weaned pigs after LPS challenge[J].The Journal of Nutrition,2012,142(11):2017-2024.
[4] LUEDDE T,KAPLOWITZ N,SCHWABE R F.Cell death and cell death responses in liver disease:mechanisms and clinical relevance[J].Gastroenterology,2014,147(4):765-783.e4.
[5] 汪龙梅.香菇多糖对脂多糖刺激的断奶仔猪肠道损伤的调控作用[D].硕士学位论文.武汉:武汉轻工大学,2018.
[6] GÜNTHER C,MARTINI E,WITTKOPF N,et al.Caspase-8 regulates TNF-α-induced epithelial necroptosis and terminal ileitis[J].Nature,2011,477(7364):335-339.
[7] 秦令祥,崔胜文,周婧琦,等.香菇多糖的提取技术研究进展[J].食品研究与开发,2018,39(18):214-218.
[8] XU W T,ZHANG F F,LUO Y B,et al.Antioxidant activity of a water-soluble polysaccharide purified from
Pteridium aquilinum[J].Carbohydrate Research,2009,344(2):217-222.
[9] ZHANG Y Y,LI S,WANG X H,et al.Advances in lentinan:isolation,structure,chain conformation and bioactivities[J].Food Hydrocolloids,2011,25(2):196-206.
[10] NISHITANI Y,ZHANG L,YOSHIDA M,et al.Intestinal anti-inflammatory activity of lentinan:influence on
IL-8 and
TNFR1 expression in intestinal epithelial cells[J].PLoS One,2013,8(4):e62441.
[11] MAO X B,HU H Y,XIAO X C,et al.Lentinan administration relieves gut barrier dysfunction induced by rotavirus in a weaned piglet model[J].Food & Function,2019,10(4):2094-2101.
[12] WANG X Y,WANG W J,WANG L M,et al.Lentinan modulates intestinal microbiota and enhances barrier integrity in a piglet model challenged with lipopolysaccharide[J].Food & Function,2019,10(1):479-489.
[13] LIANG Z E N,YUAN Z H,GUO J J,et al.
Ganoderma lucidum polysaccharides prevent palmitic acid-evoked apoptosis and autophagy in intestinal porcine epithelial cell line via restoration of mitochondrial function and regulation of MAPK and AMPK/Akt/mTOR signaling pathway[J].International Journal of Molecular Sciences,2019,20(3):478.
[14] KUCUKGUL A,ERDOGAN S.Low concentration of oleic acid exacerbates LPS-induced cell death and inflammation in human alveolar epithelial cells[J].Experimental Lung Research,2017,43(1):1-7.
[15] 陈逢.鱼油通过TLR4和NOD信号通路对脂多糖诱导的仔猪肠道、肝脏损伤和肌肉蛋白质降解的调控作用[D].硕士学位论文.武汉:武汉轻工大学,2013.
[16] LIVAK K J,SCHMITTGEN T D.Analysis of relative gene expression data using real-time quantitative PCR and 2
-ΔΔCT method[J].Methods,2001,25(4):402-408.
[17] WANG X Y,LIU Y L,LI S,et al.Asparagine attenuates intestinal injury,improves energy status and inhibits AMP-activated protein kinase signalling pathways in weaned piglets challenged with
Escherichia coli lipopolysaccharide[J].British Journal of Nutrition,2015,114(4):553-565.
[18] ZHU H L,LIU Y L,CHEN S K,et al.Fish oil enhances intestinal barrier function and inhibits corticotropin-releasing hormone/corticotropin-releasing hormone receptor 1 signalling pathway in weaned pigs after lipopolysaccharide challenge[J].British Journal of Nutrition,2016,115(11):1947-1957.
[19] VAN NEVEL C J,DECUYPERE J A,DIERICK N,et al.The influence of
Lentinus edodes (shiitake mushroom) preparations on bacteriological and morphological aspects of the small intestine in piglets[J].Archives of Animal Nutrition,2003,57(6):399-412.
[20] YOSUKE N,LING Z,MASARU Y,et al.Intestinal anti-inflammatory activity of lentinan:influence on
IL-8 and
TNFR1 expression in intestinal epithelial cells[J].PLoS One,2013,8(4):e62441.
[21] 李先根.细胞程序性坏死在脂多糖诱导的仔猪肝脏损伤中的作用[D].硕士学位论文.武汉:武汉轻工大学,2019.
[22] 于辛,陈丽香,周晓辉.程序性细胞坏死及其信号通路[J].微生物与感染,2015,10(1):61-66.
[23] 胡艳红,张凡,张楚焌,等.程序性细胞死亡形式研究进展[J].辽宁中医药大学学报,2018,20(12):85-89.
[24] WELZ P S,WULLAERT A,VLANTIS K,et al.FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation[J].Nature,2011,477(7364):330-334.
[25] LI Z Y,JIA Y Y,FENG Y,et al.Methane alleviates sepsis-induced injury by inhibiting pyroptosis and apoptosis:
in vivo and
in vitro experiments[J].Aging,2019,11(4):1226-1239.
[26] 申兵冰.程序性坏死研究进展[J].重庆医学,2018,47(3):406-409.
[27] VANDEN BERGHE T,LINKERMANN A,JOUAN-LANHOUET S,et al.Regulated necrosis:the expanding network of non-apoptotic cell death pathways[J].Nature Reviews Molecular Cell Biology,2014,15(2):135-147.
[28] 汪洋.程序性坏死信号通路在脂多糖诱导的仔猪肠道损伤中的作用[D].硕士学位论文.武汉:武汉轻工大学,2019.
[29] 王月霞,陈敏.自噬性细胞死亡及其与细胞凋亡、坏死关系研究进展[J].中国公共卫生,2016,32(10):1433-1436.
[30] LIAO A J,HU R,LI Y C,et al.The study of FTY720 on inducing apoptosis and autophagy in multiple myeloma cell line U266[J].Journal of Hematology,2011,32(32):664-667.
[31] 白晓,万多,宋晓宇.凋亡、自噬与程序性细胞坏死的信号关联网络[J].解剖科学进展,2017,23(4):430-432,435.
[32] HE C C,KLIONSKY D J.Regulation mechanisms and signaling pathways of autophagy[J].Annual Review of Genetics,2009,43:67-93.
[33] LI B C,YAO X,LUO Y H,et al.Inhibition of autophagy attenuated intestinal injury after intestinal I/R via mTOR signaling[J].Journal of Surgical Research,2019,243:363-370.
[34] JIANG Q,LIU G,CHEN J S,et al.Crosstalk between nuclear glucose-regulated protein 78 and tumor protein 53 contributes to the lipopolysaccharide aggravated apoptosis of endoplasmic reticulum stress-responsive porcine intestinal epithelial cells[J].Cellular Physiology and Biochemistry,2018,48:2441-2455.
